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Aflatoxin and toxic residue: Its influence with regard to jeopardize the chicken and tissues

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Anong Bintvihok
Praphit Klainil
Sombun Sutherat (Dept. of Livestock Development, Bangkok (Thailand). National Animal Health and Production Inst.)

Aflatoxin residues in muscles and other tissues of chickens fed aflatoxin B (200 ppb) contaminated feed containing ammonium carbonate (0.4 %) or propionic acid (0.02 %) or polyplasdon XL-DE (1000 ppm) or antitox plus (2000 ppm) for 8 weeks were examined. Histopathological changes in liver showed very severe pattern of bile duct proliferation in chicken fed aflatoxin B1 feed and aflatoxin B1-ammonium carbonate feed. However, the chicken fed aflatoxin B1-polyplasdon XL-DE and aflatoxin B1-antitox plus feed showed mild form of bile duct proliferation. SGOT activity increased in chicken fed aflatoxin B1 and the pattern of SGOT activity in chicken fed aflatoxin B1-detoxifying agents was similar and slightly dcreased to that of chicken fed aflatoxin B1 alone. However, SGPT activity rarely increased in three groups of chickens fed aflatoxin B1, aflatoxin B1-propionic acid and aflatoxin B1-ammonium carbonate. Aflatoxin residues were highest in muscles (0.117 ppb) of chickens fed aflatoxin B1 feed and they were lower in muscles (0.007, 0.004, 0.001 and 0.001 ppb) of chickens fed aflatoxin B1-polyplasdon XL-DE aflatoxin B1-antitox plus, aflatoxin B1-ammonium carbonate and aflatoxin B1-propionic acid respectively. Therefore, addition of propionic acid (0.02 %) or antiox plus (2000 ppm) or polyplasdon XL-DE (1000 ppm) or ammonium carbonate (0.4 %) to diets containing aflatoxin B1 (200 ppb) for detoxification seemed to be effective

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