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Linkage of a microsatellite marker to the canine copper toxicosis locus in Bedlington terriers.
1997
Yuzbasiyan Gurkan V. | Blanton S.H. | Cao Y. | Ferguson P. | Li J. | Venta P.J. | Brewer G.J.
Influences of breed, sex, and susceptibility to malignant hyperthermia on lipid composition of skeletal muscle and adipose tissue in swine.
1997
Hartmann S. | Otten W. | Kratzmair M. | Seewald M.J. | Iaizzo P.A. | Eichinger H.M.
Study of hereditary cerebellar degeneration in cats.
1996
Inada S. | Mochizuki M. | Izumo S. | Kuriyama M. | Sakamoto H. | Kawasaki Y. | Osame M.
Isoelectric focusing under dissociating conditions for analysis of muscle protein from clinically normal dogs and Labrador Retrievers with hereditary myopathy.
1989
Mehta J.R. | Braund K.G. | McKerrell R.E. | Toivio Kinnucan M.
Protein profiles of whole homogenates of anconeus (slow twitch) and biceps femoris (fast twitch) muscles of clinically normal dogs and of Labrador Retrievers with hereditary myopathy (HM) were resolved on flat bed polyacrylamide isoelectric-focusing gels. Three methods of sample solubilization were performed. The solubilization buffer, with high concentrations of urea, precipitated the zwitterionic detergent, but use of the buffer containing 3% NP-40, 9.2M urea, and 0.1M arginine resulted in better resolution and stability of pH gradient. Gels of anconeus muscle from clinically normal dogs contained 2 protein bands specific to anconeus muscle, whereas gels of biceps femoris muscle from clinically normal dogs contained 3 protein bands amplified in biceps femoris muscle that were barely detectable in anconeus muscle. The staining intensity of protein bands in biceps femoris muscles from Labrador Retrievers with HM was decreased, relative to controls. The quantitative analysis of peak height ratios of biceps femoris muscle revealed significant (P less than 0.05) differences between profiles of clinically normal dogs and Labrador Retrievers with HM.
Show more [+] Less [-]Diprosopus in a Holstein calf.
1987
Hishinuma M. | Kohnose M. | Takahashi Y. | Kanagawa H.
Canine storage disease characterized by hereditary progressive neurogenic muscular atrophy: breeding experiments and clinical manifestation.
1986
Inada S. | Yamauchi C. | Igata A. | Osame M. | Izumo S.
Brachygnathia in a herd of Angus cattle.
1985
Heidari M. | Vogt D.W. | Nelson S.L.
Hereditary nonspherocytic hemolytic anemia in Beagles.
1988
Maggio Price L. | Emerson C.L. | Hinds T.R. | Vincenzi F.F. | Hammond W.R.
Three Beagles with chronic anemia and reticulocytosis were studied. The dogs originated from a large breeding colony and appeared clinically normal with the exception of splenomegaly. The PCV ranged from 30 to 39% (normal, 46 to 56%), with reticulocyte indices of 2.3 to 9.9. Red blood cells were morphologically normal, and examination of marrow aspirates revealed erythroid hyperplasia. Shortened chromium-51 RBC life-spans (7.2 to 15.4 days in anemic dogs; 22.2 to 25.2 days in control dogs) documented a hemolytic anemia. Acquired causes of hemolytic anemia were ruled out. Red blood cells had normal glycolytic enzyme activities, no evidence of unstable or abnormal hemoglobin, and had altered osmotic fragility curves. The breeding of 2 anemic dogs resulted in off-spring with anemia and reticulocytosis. Polyacrylamide gel electrophoresis revealed no abnormalities in RBC membrane cytoskeletal proteins in all anemic adult dogs and in 3 offspring.
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