The effect of opportunistic microsporidian infection on the immune system in mice (an outline)
2001
Maslej, P. | Valencakova, A. | Lesnik, F. | Balent, P. | Stefkovic, M. | Oroszova, K. (University of Veterinary Medicine, Kosice (Slovak Republic)) | Halanova, M.
Opportunistic microsporidian infections introduce considerable risks, especially for patients with reduced immunity. Encephalitozoonosis as a microsporidian infection of mammals was unknown before 1995 in Slovakia. It is caused by opportunistic pathogens of Encephalitozoon spp. Active macrophages and related cells play an important role in the pathogenesis of encephalitozoonosis. Natural immunity is not based on previous individual experience of the pathogen or similar non-pathogenic microorganisms. Specific immunity is based upon the clone principle - only those cells with clones which have specific receptors react with antigens. The CD8+ lymphocyte population is soon quickly activated and increases during encephalitozoonosis. The immunosuppression during bovine papillomatosis can be couple with the appearance of opportunistic encephalitozoonosis. In the human population microsporidia have been described as opportunistic pathogens in immunocompromised patients after organ transplantations
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