Wheat (Triticum) is one of the three most important field crops used for food in the world, as well as in Latvia. The two-factorial trial was conducted at the Research and Study farm ‘Pēterlauki’, Latvia, with the aim to find out the effect of soil tillage and crop rotation on winter wheat grain yield formation. Two soil tillage systems (conventional and reduced), and three crop rotations with different winter wheat (Triticum aestivum) proportion in it (100, 67 and 25%) and different fore-crops (wheat, oilseed rape (Brassica napus ssp. oleifera), faba bean (Vicia faba)) were used. Long-term trial was started in 2009, however, the data from 2016/2017, 2017/2018 and 2018/2019 growing seasons was used for this report. The highest average wheat yield was harvested in 2017 (7.17 t haE−1), and it was significantly (p is less than 0.001) higher than yields in 2018 and 2019 (on average 6.18 t haE−1 and 5.68 t haE−1, respectively). Crop rotation with winter wheat proportion 25%, in which faba bean was wheat fore-crop, showed the highest average grain yield (7.35 t haE−1), but the lowest yield was obtained in rotation with 100% wheat proportion (on average 5.52 t haE−1). Crop rotation scheme had a significant influence on the yield (p is less than 0.001) and 1000 grain weight (p is less than 0.001). Forecrop influenced the yield (p is less than 0.001), number of spikes per 1 square m (p is less than 0.001), and number of grain per spike (p=0.008) significantly. Soil tillage did not affect the studied parameters importantly. Conditions of the trial year affected all parameters significantly.

Pyrenophora tritici-repentis is a major wheat pathogen in all wheat (Triticum spp.) growing areas worldwide. Up to date, eight P. tritici-repentis races have been described based on chlorosis, necrosis, or both symptoms caused on race differential wheat genotypes: ‘Glenlea’, 6B662, 6B365, and ‘Salamouni’. Symptom development on differential genotypes depends on the interaction of the pathogen’s necrotrophic effectors named Ptr ToxA, Ptr ToxB, and Ptr ToxC with host susceptibility genes. Ptr ToxA is encoded by the single copy gene ToxA and induces necrosis on sensitive wheat cultivars. Ptr ToxB causes chlorosis and is encoded by the multicopy gene ToxB. The Ptr ToxC is the non-proteinaceous, polar, low molecular mass molecule that also induces chlorosis, but up to date, the gene encoding this toxin is unknown. Races producing Ptr ToxA are predominant in the global Ptr population. There are several reports about new putative races of P. tritici-repentis that do not conform to the current race system, so further research is required. This study aims to collect and systematise available information about the virulence and races of P. tritici-repentis.