Mechanism of Action of the Herbicide 2‐Chloro‐3(4‐chlorophenyl) Propionate and its Methyl Ester: Interaction with Cell Responses Mediated by Auxin
1976
ANDREEV, G. K. | AMRHEIN, N.
Chlorfenprop‐methyl (the herbicidal component of BIDISIN®), and to a lesser extent the free acid, chlorfenprop, inhibit auxin mediated cell responses in coleoptiles of Avena sativa L. and Zea mays L., such as cell elongation, auxin‐uptake, ‐transport and ‐metabolism, acidification of growth media, and binding of naphthyl‐I‐acetic acid to auxin‐specific binding sites in homo‐genates of corn coleoptiles. Within a very narrow concentration range (1 to 2μM) chlorfenprop‐methyl arrests growth from 0 to 100% in sensitive cultivars. The compound displays neither auxin‐nor anti‐auxin‐activity, and only the l(—)‐enantiomer is active. The interaction of the herbicide with auxin at the level of membranes is proposed.
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