Synergistic Effects of KH-red Ginseng/chlorella on the Endurance Capacity and Immune Enhancing in Forced Swimming Tested Mice
2009
Choi, I.Y., Kyung Hee University, Seoul, Republic of Korea | Lee, J.H., Wonkwang University, Iksan, Republic of Korea | Kang, T.H., Kyung Hee University, Seoul, Republic of Korea | An, H.J., Kyung Hee University, Seoul, Republic of Korea | Kim, S.J., Kyung Hee University, Seoul, Republic of Korea | Moon, P.D., Kyung Hee University, Seoul, Republic of Korea | Kim, N.H., Kyung Hee University, Seoul, Republic of Korea | Myung, N.Y., Kyung Hee University, Seoul, Republic of Korea | Yang, D.C., Kyung Hee University, Suwon, Republic of Korea | Kang, I.C., Kyung Hee University, Seoul, Republic of Korea | Choi, Y.J., Kyung Hee University, Seoul, Republic of Korea | Kim, M.H., Chonbuk National University, Jeonju, Republic of Korea | Um, J.Y., Kyung Hee University, Seoul, Republic of Korea | Hong, S.H., Wonkwang University, Iksan, Republic of Korea | Kim, H.M., Kyung Hee University, Seoul, Republic of Korea | Jeong, H.J., Hoseo University, Asan, Republic of Korea
KH-red ginseng/chlorella (KH-RG/C) is the mixed material of the Korean red ginseng powder (Panax ginseng, 75%) and extract of Chlorella vulgaris (25%). To evaluate the effects of KH-RG/C on endurance capacity and immune regulation, the forced swimming test (FST) was conducted. The immobility time in the FST was significantly decreased in KH-RG/C treated group compared with the DW-treated group at the 3 and 10 days, respectively. In the analysis of the blood biochemical parameters, KH-RG/C treatment significantly increased the glucose level. However, the lactic dehydrogenase level decreased. Although KH-RG/C increased aspartate aminotransferase, it was not different significantly. And KH-RG/C had no affects in the alanine aminotransferase, and blood urea nitrogen levels. In splenocytes and macrophages, KH-RG/C also did not affect the interleukin (IL)-2, IL-4, and IL-12 production. These results suggest that KH-RG/C may influence to immune regulation through increasing the physical endurance capacity without effect in activation of immune cells.
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