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Virulence of Pyrenophora tritici-repentis: a minireview
2021
Kaneps, J., Latvia Univ. of Life Sciences and Technologies, Jelgava (Latvia) | Bankina, B., Latvia Univ. of Life Sciences and Technologies, Jelgava (Latvia) | Morocko-Bicevska, I., Latvia Univ. of Life Sciences and Technologies, Jelgava (Latvia);Institute of Horticulture, Ceriņi, Krimūna Parish, Dobele Municipality (Latvia)
Pyrenophora tritici-repentis is a major wheat pathogen in all wheat (Triticum spp.) growing areas worldwide. Up to date, eight P. tritici-repentis races have been described based on chlorosis, necrosis, or both symptoms caused on race differential wheat genotypes: ‘Glenlea’, 6B662, 6B365, and ‘Salamouni’. Symptom development on differential genotypes depends on the interaction of the pathogen’s necrotrophic effectors named Ptr ToxA, Ptr ToxB, and Ptr ToxC with host susceptibility genes. Ptr ToxA is encoded by the single copy gene ToxA and induces necrosis on sensitive wheat cultivars. Ptr ToxB causes chlorosis and is encoded by the multicopy gene ToxB. The Ptr ToxC is the non-proteinaceous, polar, low molecular mass molecule that also induces chlorosis, but up to date, the gene encoding this toxin is unknown. Races producing Ptr ToxA are predominant in the global Ptr population. There are several reports about new putative races of P. tritici-repentis that do not conform to the current race system, so further research is required. This study aims to collect and systematise available information about the virulence and races of P. tritici-repentis.
显示更多 [+] 显示较少 [-][Clinical investigations of parr's fin necrosis at Latvian fish farms]
2001
Medne, R. (Latvia Univ. of Agriculture, Jelgava (Latvia). Inst. of Food and Environmental Hygiene)
Salmon (Salmo salar L.) is grown and released in the estuaries in Latvia from which it goes to the feeding areas in the Baltic Sea. The research summaries data about clinical characteristics of fin necrosis and its prevalence in Latvian hatcheries before the smolt stage (complicated physiological, biochemical and morphophysiological changes resulting in migration flight). Clinically expressed fin necrosis was observed in 92.7-97.4% cases.
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