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Intranasal administration of Pasteurella multocida toxin in a challenge-exposure model used to induce subclinical signs of atrophic rhinitis in pigs.
1994
Diemen P.M. van | Jong M.F. de | Vries Reilingh G. de | Hel P. van der | Schrama J.W.
A challenge-exposure model was developed for dose-dependent induction of subclinical (moderate) atrophic rhinitis (AR) in conventionally raised Dutch Landrace and Large White pigs, about 4 weeks old. Under favorable climatic and housing conditions, pigs were intranasally challenge-exposed with Pasteurella multocida-derived toxin (Pm-T) 3 days after pretreatment by inoculation with 1% acetic acid. Pigs were challenge-exposed with 1 of the following Pm-T doses: 0 (control), 5, 13, 20, or 40 microgram of Pm-T/ml of phosphate-buffered saline solution (PBSS), 0.5 ml/ nostril/d on 3 consecutive days. Five weeks after challenge exposure, subclinical moderate) AR status was defined as intermediate conchal atrophy (grade 2 for ventral conchae on a 0 to 4 scale and grade 1 or 2 for dorsal conchae on a 0 to 3 scale, respectively) and perceptible difference in change in brachygnathia superior (CBS) between control and challenge-exposed pigs between the beginning and end of the study. All Pm-T-exposed pigs had nasal damage that was dose-dependent. The higher Pm-T doses resulted in higher ventral conchae atrophy and dorsal conchae atrophy scores. The CBS increased with applied Pm-T dose, resulting in significant (P < 0.05) differences between controls (3.88 mm) and the 13-, 20-, and 40-microgram Pm-T-treated groups (7.77, 6.58, and 7.98 mm, respectively). In response to the applied dose, weight gain per week for Pm-T-exposed pigs was lower than that of controls after week 3 (P < 0.01). Difference from controls was 32, 54, 52, and 96 g/d/pig for 5-, 13-, 20-, and 40-microgram Pm-T-treated groups respectively, in the last 2 weeks. For Dutch Landrace and Large White pigs, intranasally administered Pm-T mimicked the pathogenic effect of in vivo infection with toxigenic Pm strains. The optimal model to induce subclinical AR appeared to be 13 microgram of Pm-T/ml (0.5 ml/nostril/d) on 3 consecutive days.
Mostrar más [+] Menos [-]Administration of ochratoxin A and T-2 toxin to growing swine.
1994
Harvey R.B. | Kubena L.F. | Elissalde M.H. | Rottinghaus G.E. | Corrier D.E.
Effects of dietary ochratoxin A (OA) and T-2 toxin, fed singly and in combination, were evaluated in growing crossbred pigs. Thirty-six barrows (3 replicates of 3 for each of 4 treatment groups, mean body weight, 18.0 kg) were fed: 0 mg of OA and 0 mg of T-2/kg of feed (control); 2.5 mg of OA/kg of feed; 8.0 mg of T-2/kg of feed; or 2.5 mg of OA plus 8.0 mg of T-2/kg of feed for 30 days. Production performance, serum biochemical, hematologic, immunologic, and pathologic evaluations were made. Body weight and body weight gain were decreased by all toxin treatments, but the combination toxin treatment reduced weight gain more than did either of the toxins administered singly and could be considered additive. Liver weight was decreased by combination treatment, whereas kidney weight was increased by OA treatment. Ochratoxin decreased serum cholesterol, inorganic phosphorus, and alkaline phosphatase values; reduced mean cell volume, hemoglobin concentration, and macrophage phagocytosis; and increased creatinine and total protein values. Consumption of T-2 toxin reduced hemoglobin and serum alkaline phosphatase values. The combination treatment decreased serum cholesterol, gamma-glutamyltransferase, alkaline phosphatase, mean cell volume, hematocrit, and hemoglobin values, as well as lymphoblastogenesis and phagocytosis, and increased serum nine concentration. We concluded that OA and T-2, singly or in combination, can affect clinical performance, serum biochemical, hematologic, and immunologic values, and organ weights of growing barrows. Although some analytes were affected more by the combination than by either toxin alone, the interactions could best be described as additive, not synergistic.
Mostrar más [+] Menos [-]Pathological studies on exudative epidermitis in experimentally infected pigs-(1)-Macroscopical and histopathological observations.
1994
Oh K.S. | Lee C.S.
Identification of Staphylococcus hyicus subsp hyicus of swine and poultry origin by API STAPH-IDENT system.
1994
Park C.K.
Restriction endonuclease analysis of a porcine isolate of bovine herpesvirus type I.
1994
Varady E. | Tuboly T. | Derbyshire J.B.
Studies on Pasteurella multocida isolated from pneumonic lungs of slaughter pigs
1994
Ahn, B.C. | Kim, B.H. (Kyungpook National University, Taegu (Korea Republic). College of Veterinary Medicine) | Cho, K.H. (Kyungpook Animal Health Experiment Institute, Taegu (Korea Republic))
Isolation of porcine reproductive and respiratory syndrome virus (PRRSV) in Korea
1994
Kweon, C.H. | Kwon, B.J. | Lee, H.J. | Cho, J.J. | Hwang, E.K. | Shin, J.H. | Yoon, Y.D. | Kang, Y.B. | An, S.H. (Rural Development Administration, Anyang (Korea Republic). Veterinary Research Institute) | Kim, Y.H. (Bayer Veterinary Medical Research Institute, Anyang (Korea Republic)) | Huh, W. (Daesung Laboratory, Anyang (Korea Republic)) | Jun, M.H. (Chungnam National University, Taejon (Korea Republic). College of Veterinary Medicine) | Wensvoort, G. (Central Veterinary Institute, Lelystad (Netherlands))
Studies on the distribution of intestinal microflora and characterization of Bifidobacterium isolated from the intestine of domestics animals
1994
Lee, W.K. (Chungbuk National University, Cheongju (Korea Republic). College of Veterinary Medicine)
Application of immunohistochemical technique for diagnosis of porcine epidemic diarrhea
1994
Park, N.Y. | Cho, K.O. (Chonnam National University, Kwangju (Korea Republic). College of Veterinary Medicine)
Enhancement of cell-mediated immunity by administration of plasma protein in pigs-(1)-Proportion of leukocyte subpopulations and cells expressing adhesion molecules in peripheral blood
1994
Yang, C.K. (Thaihan Industry Company, Seoul (Korea Republic). Veterinary Diagnostic Laboratory) | Kim, S.J. (Konkuk University, Seoul (Korea Republic). Department of Veterinary Medicine) | Moon, J.S. | Jung, S.C. | Park, Y.H. (Rural Development Administration, Anyang (Korea Republic). Veterinary Research Institute)